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All stimulants act to boost the extracellular concentrations of dopamine, norepinephrine, and serotonin. Stimulant medication can block the transport of these neurotransmitters.
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This discrepancy isn't shared with other dopamine reuptake inhbitors like bupropion, sibutramine, mazindol or tesofensine, that have identical or bigger potencies than copyright as dopamine reuptake inhibitors. These findings have evoked a hypothesis that copyright might also function to be a so-identified as "DAT inverse agonist" or "unfavorable allosteric modifier of DAT" leading to dopamine transporter reversal, and subsequent dopamine launch to the synaptic cleft from the axon terminal in a very manner much like but unique from amphetamines.[116]
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